CANCER GENETICS
Bcb30-009	A p53-dependent FBXO44-RAD18 axis limits mutagenesis by terminating translesion DNA synthesis
	Quote:	These findings define a p53-FBXO44-RAD18 regulatory axis that limits mutagenic TLS and helps safeguard genome integrity after replication stress.
	Article:	https://www.biorxiv.org/content/10.64898/2026.07.27.739831v1
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	Search Authors:	Alessio Butera Sabrina Caporali Francesco Capradossi Paul Smith Nino Tabagari Nora Nikoloska Olga Mayans Ana Janic Andreas J Gruber Vincenzo DAngiolella Ivano Amelio,
PRIME AGING GENETICS
Bgn30-005	Genome-wide multi-layered epigenomic profiling across human aging
	Quote:	Collectively, our integrative genome-wide analysis revealed only limited association between age-associated epigenetic alterations in DNA methylation, chromatin accessibility, and histone modifications, arguing against a broadly coordinated remodeling of the aging epigenome.
	Article:	https://www.biorxiv.org/content/10.64898/2026.07.27.740934v1
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	Search Authors:	Mara Steiger Rene Kruger Mina Shaigan Deepika Puri Giulia Fornero Hannes Klump Alexander Meissner Ivan Gesteira Costa Filho Helene Kretzmer Wolfgang Wagner,
GENETICS
Bmo30-009	New histone deposition recruits the DNA methylation maintenance machinery at sites of DNA damage repair
	Quote:	Functionally, we reveal the importance of the DNA methylation maintenance machinery for the transcriptional response to UV damage and sustained cell proliferation. This work provides a comprehensive picture of DNA methylation control mechanisms following DNA damage, with important implications for our understanding of human diseases with an altered methylome.
	Article:	https://www.biorxiv.org/content/10.64898/2026.07.27.740930v1
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	Search Authors:	Margherita Mori Sandra Piquet Lea Girard Laure Ferry Kosuke Yamaguchi Melina Farshchi Elouan Bethouel Olivier Kirsh Magali Hennion Pierre-Antoine Defossez Sophie E Polo,
CANCER GENETICS
Sd301-001	Why the Same DNA Damage Causes Cancer in Some People but Not Others/a
	Quote:	A controlled mouse study has provided direct evidence that inherited genetics can steer how cancer begins and evolves after DNA damage. The discovery could eventually help doctors better predict cancer risk and tailor screening and treatments... Although the tumors frequently activated the same broad pathway, they did not all evolve in the same way. The specific driver mutations that appeared depended on the inherited genetics of each mouse.
	Article:	https://www.sciencedaily.com//releases/2026/07/260727012123.htm
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Javilk's Research Links Newsletter for Week 30
Updated 08/01/2026-01:13:17 AM
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Bcb30-009

A p53-dependent FBXO44-RAD18 axis limits mutagenesis by terminating translesion DNA synthesis

Bgn30-005

Genome-wide multi-layered epigenomic profiling across human aging

Bmo30-009

New histone deposition recruits the DNA methylation maintenance machinery at sites of DNA damage repair

Sd301-001

Why the Same DNA Damage Causes Cancer in Some People but Not Others/a