Research
Topics:
INDEX
AI-chat 3
MCS 1
PRIME 28
PRIO 3
aging 7
brain 9
brainwave 1
cancer 8
climate 3
cognition 4
covid-19 2
crime 5
culture 3
dental 1
diabetes 3
drugs 2
economy 2
energy 3
exercise 1
eyes 5
gender 1
genetics 5
gut 1
heart 2
immune 5
language 1
metabolism 1
microbiome 2
nutrition 5
obesity 1
pain 1
physical 1
politics 8
population 6
practice 1
psychology 1
quantum 1
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rights 1
sleep 2
tech 1
vaccine 2
war-confl
war-cyber 1
 
Scanned: 3298
Evaluated: 234
Selected: 59

 
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Bbc42-007

Holistic Design of Selective Photoenzymes with Genetically Programmed Sensitizers

Article: https://www.biorxiv.org/content/10.64898/2026.10.16.759720v1
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Search Authors: Florence J. Hardy Rebecca Crawshaw Christophe Dider Anna R Kohn Francis J. Gallois Anna Lauko Samuel J. Pellock Indrek Kalvet Anthony P. Green David Baker, 
    PRIME    PRIO    CANCER    NUTRITION   

Bcb42-004

Ascorbate is a physiological tumor suppressor metabolite in leukemia

Quote: Ascorbate restoration in mice with established leukemias extended survival. Hematopoietic-specific ascorbate depletion caused leukemia in collaboration with TET2 loss and was sufficient to cause leukemia with high penetrance even in the absence of other engineered mutations. These results identify ascorbate as a physiological tumor suppressor metabolite in blood cancers.
Article: https://www.biorxiv.org/content/10.64898/2026.10.20.760526v1
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Search Authors: Tobias Wijshake Joseph Rose span classnlm-suffixIII Ji Hyung Jun Edward O. Kwarteng Xiaotong Yang Jacob Zielke Swetha Mahesula Li Li Stephen S. Chung Stefano Comazzetto Sean J. Morrison Weina Chen Michalis Agathocleous, 
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Bcb42-005

Glycoconjugates tune the viscoelastic signature of mammary tumor progression

Quote: Our work establishes increased viscoelasticity as a defining mechanical feature of breast cancer progression, driven in part by the accumulation of glycoconjugates. These findings provide a fuller picture of tumor mechanical evolution and a benchmark for recapitulating matrix viscoelasticity in engineered models of breast cancer.
Article: https://www.biorxiv.org/content/10.64898/2026.10.20.760544v1
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Search Authors: Abhishek Sharma Jane A Baude Maya Papez Marissa Gionet-Gonzales Ryan S Stowers, 
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Bcb42-006

Temporal Dynamics of SMARCA4 Loss Define Its Role in Myogenic Differentiation of Alveolar Rhabdomyosarcoma

Article: https://www.biorxiv.org/content/10.64898/2026.10.20.760536v1
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Search Authors: Charles Keller Narendra Bharathy Elangovan Charity Conlin Jennings Emma Lashyro Jiaojiao Wang Gillian St. John, 
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Bcb42-007

Cytoplasmic localization of 5hmC reveals RNA hydroxymethylation in breast cancer

Article: https://www.biorxiv.org/content/10.64898/2026.10.21.760667v1
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Search Authors: Jinrui Wen Lisa Del Bel Belluz Silvia Ottaviani Fotini Poyia Hemeng Yu Saachi Sachdev Liliane El Eid Katherine Garner Krina Mistry Emily Hayes Nina Moderau Pramodh Chitral Chandrasinghe Justin Stebbing Biancastella Cereser, 
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Bcb42-008

Oncogenic KRAS-MAPK signaling sustains a CD73 dependent therapeutic vulnerability in pancreatic cancer

Article: https://www.biorxiv.org/content/10.64898/2026.10.20.760559v1
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Search Authors: Khadija Turabi Urvinder Kaur Sardarni Katherine E. Ankenbauer Sabrina M. Torbit Vidhi Chandra Christopher P. Taranto Shwetapadma Dash Audrey Meade Amelia T. Cephas Kelsey A. Klute Marina Pasca di Magliano Florencia McAllister Thomas McFall Kathleen E DelGiorno Jennifer M. Bailey-Lundberg, 
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Bcb42-010

Testis-Specific MAGEA3/6 Regulate Glycolysis to Promote Metabolic Flexibility and Reveal Therapeutic Vulnerabilities in Pancreatic Cancer Cells

Quote: Given that MAGEA3/6 are physiologically expressed primarily in the male germline during early spermatogenesis, a process highly sensitive to metabolic and oxidative stress, these findings also raise important questions about their physiological roles in protecting developing germ cells and maintaining metabolic and redox homeostasis. Together, our findings recognize MAGEA3/6 as regulators of metabolic plasticity in PDAC and highlight how understanding their molecular functions in both spermatogenesis and cancer may uncover previously unrecognized metabolic vulnerabilities and potential therapeutic opportunities.
Article: https://www.biorxiv.org/content/10.64898/2026.10.20.760599v1
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Search Authors: Sima Tozandehjani Vesna Jurjevic Denis Stepihar Maria Camila Hoyos Sanchez Sara Uhan Jerneja Koren Rebecca R. Florke Gee Juan Sebastian Solano-Gutierrez Emily Ballard Jesenia M. Perez Menglin Jiang Lara Snoj Barbara Breznik Vittori Milos Vittori Patrick Ryan Potts Klementina Fon Tacer, 
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Sd421-003

Why Painful Arthritis Can Return Stronger After Cancer Treatment

Article: https://www.sciencedaily.com//releases/2026/10/261018205055.htm
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  Javilk's Research Links Newsletter for Week 42
Updated 10/24/2026-02:39:34 AM
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